[AlinB]

În prealabil postat de bluester

Inainte de fiecare transfuzie, primitorului i se fac teste pentru determinarea grupei sangelui si rh-ului; si in functie de acestea doua, primitorul va primi sange compatibil cu al sau. Deci, daca se respecta protocoalele, transfuzia nu face nici un rau.
Nu va luati dupa martorii lui iehova... ei manipuleaza intr-un mod grosolan.

Da, exista ceea ce spune respectivul dar se intampla foarte rar:

https://en.wikipedia.org/wiki/Blood_...dverse_effects

Acute hemolytic reactions are defined according to Serious Hazards of Transfusion (SHOT) as "fever and other symptoms/signs of haemolysis within 24 hours of transfusion; confirmed by one or more of the following: a fall of Hb, rise in lactate dehydrogenase (LDH), positive direct antiglobulin test (DAT), positive crossmatch" [26] This is due to destruction of donor red blood cells by preformed recipient antibodies. Most often this occurs due to clerical errors or improper ABO blood typing and crossmatching resulting in a mismatch in ABO blood type between the donor and the recipient. Symptoms include fever, chills, chest pain, back pain, hemorrhage, increased heart rate, shortness of breath, and rapid drop in blood pressure. When suspected, transfusion should be stopped immediately, and blood sent for tests to evaluate for presence of hemolysis. Treatment is supportive. Kidney injury may occur due to the effects of the hemolytic reaction (pigment nephropathy). In the USA in 2011 there were 48 episodes of acute hemolysis due to a mismatch in ABO blood type (1 in 495,207 blood components transfused) and 168 episodes of hemolysis due to other causes (1 in 124,525 blood components transfused).[27] In the UK in 2014 there were 18 episodes of hemolysis due to other causes (1 in 147,972 blood components transfused).[26]
Delayed hemolytic reactions occur more than 24 hours after a transfusion,[26] and occur more frequently (1 in 20,569 blood components transfused in the USA in 2011).[27] They are due to the same mechanism as in acute hemolytic reactions. However, the consequences are generally mild and a great proportion of patients may not have symptoms. However, evidence of hemolysis and falling hemoglobin levels may still occur. Treatment is generally not needed, but due to the presence of recipient antibodies, future compatibility may be affected.
Febrile nonhemolytic reactions are the most common type of blood transfusion reaction and occur due to the release of inflammatory chemical signals released by white blood cells in stored donor blood. This type of reaction occurs in about 7% of transfusions. Fever is generally short lived and is treated with antipyretics, and transfusions may be finished as long as an acute hemolytic reaction is excluded. This is a reason for the now-widespread use of leukoreduction – the filtration of donor white cells from red cell product units.[14]
Allergic reactions may occur when the recipient has preformed antibodies to certain chemicals in the donor blood, and does not require prior exposure to transfusions. Symptoms include hives, itching, low blood pressure, and respiratory distress which may lead to anaphylactic shock. Treatment is the same as for any other type 1 hypersensitivity reactions and includes administering intramuscular epinephrine, glucocorticoids, antihistamines, medications to keep the blood pressure from dropping, and mechanical ventilation if needed. A small population (0.13%) of patients are deficient in the immunoglobulin IgA, and upon exposure to IgA-containing blood, may develop an anaphylactic reaction.
Posttransfusion purpura is a rare complication that occurs after transfusion of red cells or platelets and is associated with the presence antibodies in the patient's blood directed against the HPA (human platelet antigen) systems (only one case was reported in the UK in 2014).[26] Recipients who lack this protein develop sensitization to this protein from prior transfusions or previous pregnancies, and develop thrombocytopenia about 5 to 12 days after subsequent transfusions. Treatment is with intravenous immunoglobulin.[28]
Transfusion-associated acute lung injury (TRALI) is a syndrome of acute respiratory distress, often associated with fever, non-cardiogenic pulmonary edema, and hypotension, which may occur as often as 1 in 2000 transfusions.[29] Symptoms can range from mild to life-threatening, but most patients recover fully within 96 hours, and the mortality rate from this condition is less than 10%.[30] Although the cause of TRALI is not clear, it has been consistently associated with anti-HLA antibodies. Because these types of antibodies are commonly formed during pregnancy, several transfusion organisations have decided to use only plasma from men for transfusion.[31] TRALI is typically associated with plasma components rather than packed red blood cells (RBCs), though there is some residual plasma in RBC units.[31]

Manipularea provne de la faptul ca se generalizeaza, absurd, il lasa pe ala sa moara cu zile dupa "logica": mai bine sa moara sigur decat sa moara eventual prin transfuzie cu o sansa de 1 la 100.000.

Minciuna e unul din simptomele fratiei cu "tatal minciunii" iar asta e una dintre cele mai penibile tentative.